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‘Trapped re-entry’ as source of acute focal atrial arrhythmias. / De Coster, Tim; Teplenin, Alexander S.; Feola, Iolanda и др.
в: Cardiovascular research, Том 120, № 3, 14.03.2024, стр. 249-261.

Результаты исследований: Вклад в журнал › Статья › Рецензирование

Harvard

De Coster, T, Teplenin, AS, Feola, I, Bart, CI, Ramkisoensing, AA, Den Ouden, BL, Ypey, DL, Trines, SA, Panfilov, AV, Zeppenfeld, K, De Vries, AAF & Pijnappels, DA 2024, '‘Trapped re-entry’ as source of acute focal atrial arrhythmias', Cardiovascular research, Том. 120, № 3, стр. 249-261. https://doi.org/10.1093/cvr/cvad179

APA

De Coster, T., Teplenin, A. S., Feola, I., Bart, C. I., Ramkisoensing, A. A., Den Ouden, B. L., Ypey, D. L., Trines, S. A., Panfilov, A. V., Zeppenfeld, K., De Vries, A. A. F., & Pijnappels, D. A. (2024). ‘Trapped re-entry’ as source of acute focal atrial arrhythmias. Cardiovascular research, 120(3), 249-261. https://doi.org/10.1093/cvr/cvad179

Vancouver

De Coster T, Teplenin AS, Feola I, Bart CI, Ramkisoensing AA, Den Ouden BL и др. ‘Trapped re-entry’ as source of acute focal atrial arrhythmias. Cardiovascular research. 2024 март 14;120(3):249-261. doi: 10.1093/cvr/cvad179

Author

De Coster, Tim ; Teplenin, Alexander S. ; Feola, Iolanda и др. / ‘Trapped re-entry’ as source of acute focal atrial arrhythmias. в: Cardiovascular research. 2024 ; Том 120, № 3. стр. 249-261.

BibTeX

@article{a53392e4a120462eb4c23facfc691f2d,
title = "{\textquoteleft}Trapped re-entry{\textquoteright} as source of acute focal atrial arrhythmias",
abstract = "Diseased atria are characterized by functional and structural heterogeneities, adding to abnormal impulse generation and propagation. These heterogeneities are thought to lie at the origin of fractionated electrograms recorded during sinus rhythm (SR) in atrial fibrillation (AF) patients and are assumed to be involved in the onset and perpetuation (e.g. by re-entry) of this disorder. The underlying mechanisms, however, remain incompletely understood. Here, we tested whether regions of dense fibrosis could create an electrically isolated conduction pathway (EICP) in which re-entry could be established via ectopy and local block to become {\textquoteleft}trapped{\textquoteright}. We also investigated whether this could generate local fractionated electrograms and whether the re-entrant wave could {\textquoteleft}escape{\textquoteright} and cause a global tachyarrhythmia due to dynamic changes at a connecting isthmus.",
author = "{De Coster}, Tim and Teplenin, {Alexander S.} and Iolanda Feola and Bart, {Cindy I.} and Ramkisoensing, {Arti A.} and {Den Ouden}, {Bram L.} and Ypey, {Dirk L.} and Trines, {Serge A.} and Panfilov, {Alexander v} and Katja Zeppenfeld and {De Vries}, {Antoine A. F.} and Pijnappels, {Dani{\"e}l A.}",
note = "This work was supported by The European Research Council (ERC Starting grant 716509) and the Netherlands Organization for Scientific Research (NWO Vidi grant 91714336), both to D.A.P. In addition A.V.P. was supported by the Russian Ministry of Science and Higher Education (grant 075-15-2022-304).",
year = "2024",
month = mar,
day = "14",
doi = "10.1093/cvr/cvad179",
language = "English",
volume = "120",
pages = "249--261",
journal = "Cardiovascular research",
issn = "0008-6363",
publisher = "Oxford University Press",
number = "3",

}

RIS

TY - JOUR

T1 - ‘Trapped re-entry’ as source of acute focal atrial arrhythmias

AU - De Coster, Tim

AU - Teplenin, Alexander S.

AU - Feola, Iolanda

AU - Bart, Cindy I.

AU - Ramkisoensing, Arti A.

AU - Den Ouden, Bram L.

AU - Ypey, Dirk L.

AU - Trines, Serge A.

AU - Panfilov, Alexander v

AU - Zeppenfeld, Katja

AU - De Vries, Antoine A. F.

AU - Pijnappels, Daniël A.

N1 - This work was supported by The European Research Council (ERC Starting grant 716509) and the Netherlands Organization for Scientific Research (NWO Vidi grant 91714336), both to D.A.P. In addition A.V.P. was supported by the Russian Ministry of Science and Higher Education (grant 075-15-2022-304).

PY - 2024/3/14

Y1 - 2024/3/14

N2 - Diseased atria are characterized by functional and structural heterogeneities, adding to abnormal impulse generation and propagation. These heterogeneities are thought to lie at the origin of fractionated electrograms recorded during sinus rhythm (SR) in atrial fibrillation (AF) patients and are assumed to be involved in the onset and perpetuation (e.g. by re-entry) of this disorder. The underlying mechanisms, however, remain incompletely understood. Here, we tested whether regions of dense fibrosis could create an electrically isolated conduction pathway (EICP) in which re-entry could be established via ectopy and local block to become ‘trapped’. We also investigated whether this could generate local fractionated electrograms and whether the re-entrant wave could ‘escape’ and cause a global tachyarrhythmia due to dynamic changes at a connecting isthmus.

AB - Diseased atria are characterized by functional and structural heterogeneities, adding to abnormal impulse generation and propagation. These heterogeneities are thought to lie at the origin of fractionated electrograms recorded during sinus rhythm (SR) in atrial fibrillation (AF) patients and are assumed to be involved in the onset and perpetuation (e.g. by re-entry) of this disorder. The underlying mechanisms, however, remain incompletely understood. Here, we tested whether regions of dense fibrosis could create an electrically isolated conduction pathway (EICP) in which re-entry could be established via ectopy and local block to become ‘trapped’. We also investigated whether this could generate local fractionated electrograms and whether the re-entrant wave could ‘escape’ and cause a global tachyarrhythmia due to dynamic changes at a connecting isthmus.

UR - http://www.scopus.com/inward/record.url?partnerID=8YFLogxK&scp=85188178652

UR - https://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=tsmetrics&SrcApp=tsm_test&DestApp=WOS_CPL&DestLinkType=FullRecord&KeyUT=001126965900001

U2 - 10.1093/cvr/cvad179

DO - 10.1093/cvr/cvad179

M3 - Article

VL - 120

SP - 249

EP - 261

JO - Cardiovascular research

JF - Cardiovascular research

SN - 0008-6363

IS - 3

ER -

ID: 55296779